Which was largely absent in vaccine recipients, was a characteristic of immune responses in COVID-19 patients.” The observed sharp overexpression.
Of cytotoxic genes in peripheral T cells and innate-like lymphocytes in patients, but not in vaccinated participants, was probably caused by increased interferon signaling.
The research further states that “Analysis of B and T cell receptor repertoires revealed that while the majority of clonal B and T cells.
In COVID-19 patients were effector cells, in vaccine recipients clonally expanded cells were primarily circulating memory cells.”
Simply put, this indicates that although memory response necessitates a slower-responding adaptive mode, effector cells initiate a faster and more robust innate response.
